Homozygosity for CAG mutation in Huntington disease is associated with a more severe clinical course.
Mechanisms of pure alexia: spatially based impairment, letter identification deficit, or both?
Integrating fetal neural transplants into a therapeutic strategy: the example of Huntington's disease.
Time to imagine space: a chronometric exploration of representational neglect.
Cognitive impairment related to apathy in early Huntington's disease.
Electrophysiological deterioration over time in patients with Huntington's disease.
Severity of dysfluency correlates with basal ganglia activity in persistent developmental stuttering.
Severe deficiency of the fatty acid amide hydrolase (FAAH) activity segregates with the Huntington's disease mutation in peripheral lymphocytes.
Longitudinal evaluation of "presymptomatic" carriers of Huntington's disease.
Different neurophysiological mechanisms underlying word and rule extraction from speech.